Cholesterol secoaldehyde induces apoptosis in H9c2 cardiomyoblasts through reactive oxygen species involving mitochondrial and death receptor pathways. [electronic resource]
Producer: 20091207Description: 548-58 p. digitalISSN:- 1873-4596
- Aldehydes -- chemistry
- Apoptosis -- drug effects
- Caspases -- metabolism
- Cell Line
- Cholestanones -- pharmacology
- Cholesterol -- chemistry
- Cytochromes c -- metabolism
- Dose-Response Relationship, Drug
- Drug Evaluation, Preclinical
- Humans
- Membrane Potential, Mitochondrial -- drug effects
- Mitochondria, Heart -- drug effects
- Models, Biological
- Myocytes, Cardiac -- drug effects
- Oxidative Stress -- drug effects
- Reactive Oxygen Species -- pharmacology
- Receptors, Death Domain -- metabolism
- Secosteroids -- pharmacology
- Signal Transduction -- drug effects
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
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