Bacterial endotoxin enhances colorectal cancer cell adhesion and invasion through TLR-4 and NF-kappaB-dependent activation of the urokinase plasminogen activator system. [electronic resource]
Producer: 20090602Description: 1589-602 p. digitalISSN:- 1532-1827
- Amiloride -- pharmacology
- Antibodies -- pharmacology
- Bacterial Toxins -- pharmacology
- Caco-2 Cells
- Carcinoma -- metabolism
- Cell Adhesion -- drug effects
- Cell Movement -- drug effects
- Colorectal Neoplasms -- metabolism
- Humans
- Lipopolysaccharides -- pharmacology
- NF-kappa B -- metabolism
- Neoplasm Invasiveness
- Receptors, Urokinase Plasminogen Activator -- antagonists & inhibitors
- Toll-Like Receptor 4 -- antagonists & inhibitors
- Tumor Cells, Cultured
- Urokinase-Type Plasminogen Activator -- metabolism
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Publication Type: Journal Article
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