KCC2 expression promotes the termination of cortical interneuron migration in a voltage-sensitive calcium-dependent manner. [electronic resource]
Producer: 20090507Description: 53-71 p. digitalISSN:- 1097-4199
- Age Factors
- Animals
- Animals, Newborn
- Bicuculline -- analogs & derivatives
- Calcium -- metabolism
- Calcium Channel Blockers -- pharmacology
- Calcium Channels, L-Type -- physiology
- Cell Movement -- drug effects
- Cerebral Cortex -- cytology
- Electroporation -- methods
- Embryo, Mammalian
- Female
- GABA Agents -- pharmacology
- Gene Expression Regulation -- drug effects
- Glutamic Acid -- pharmacology
- Green Fluorescent Proteins -- genetics
- Homeodomain Proteins -- genetics
- Interneurons -- physiology
- LIM-Homeodomain Proteins
- Membrane Potentials -- drug effects
- Mice
- Mice, Inbred BALB C
- Mice, Transgenic
- Microtubule-Associated Proteins -- metabolism
- Models, Biological
- Muscimol -- pharmacology
- Nerve Tissue Proteins -- genetics
- Nifedipine -- pharmacology
- Organ Culture Techniques
- Pregnancy
- Quinoxalines -- pharmacology
- RNA, Small Interfering -- pharmacology
- Receptors, N-Methyl-D-Aspartate -- antagonists & inhibitors
- Sequence Deletion
- Symporters -- antagonists & inhibitors
- Transcription Factors
- Valine -- analogs & derivatives
- gamma-Aminobutyric Acid -- pharmacology
- omega-Conotoxin GVIA -- pharmacology
- K Cl- Cotransporters
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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