Sar1-GTPase-dependent ER exit of KATP channels revealed by a mutation causing congenital hyperinsulinism. [electronic resource]
Producer: 20090813Description: 2400-13 p. digitalISSN:- 1460-2083
- Amino Acid Motifs
- Amino Acid Sequence
- Cell Line
- Congenital Hyperinsulinism -- genetics
- Endoplasmic Reticulum -- genetics
- Humans
- Insulin -- metabolism
- Insulin Secretion
- KATP Channels -- genetics
- Molecular Sequence Data
- Monomeric GTP-Binding Proteins -- genetics
- Mutation, Missense
- Potassium Channels, Inwardly Rectifying -- chemistry
- Protein Binding
- Protein Transport
- Sequence Alignment
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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