ERK phosphorylation mediates sildenafil-induced myocardial protection against ischemia-reperfusion injury in mice. [electronic resource]
Producer: 20090619Description: H1236-43 p. digitalISSN:- 0363-6135
- Animals
- Cardiovascular Agents -- pharmacology
- Cyclic GMP-Dependent Protein Kinases -- metabolism
- Cyclic Nucleotide Phosphodiesterases, Type 5 -- metabolism
- Disease Models, Animal
- Enzyme Activation
- Flavonoids -- pharmacology
- Glycogen Synthase Kinase 3 -- metabolism
- Glycogen Synthase Kinase 3 beta
- Hemodynamics -- drug effects
- Male
- Mice
- Mice, Inbred ICR
- Mitogen-Activated Protein Kinase 1 -- antagonists & inhibitors
- Mitogen-Activated Protein Kinase 3 -- antagonists & inhibitors
- Myocardial Contraction -- drug effects
- Myocardial Infarction -- enzymology
- Myocardial Reperfusion Injury -- enzymology
- Myocardium -- enzymology
- Nitric Oxide Synthase Type II -- metabolism
- Nitric Oxide Synthase Type III -- metabolism
- Phosphodiesterase 5 Inhibitors
- Phosphodiesterase Inhibitors -- pharmacology
- Phosphorylation
- Piperazines -- pharmacology
- Protein Kinase Inhibitors -- pharmacology
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Purines -- pharmacology
- Signal Transduction -- drug effects
- Sildenafil Citrate
- Sulfones -- pharmacology
- Time Factors
- bcl-2-Associated X Protein -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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