Rosiglitazone and PPAR-gamma overexpression protect mitochondrial membrane potential and prevent apoptosis by upregulating anti-apoptotic Bcl-2 family proteins. [electronic resource]
Producer: 20090514Description: 58-71 p. digitalISSN:- 1097-4652
- 3-Phosphoinositide-Dependent Protein Kinases
- Anilides -- pharmacology
- Animals
- Apoptosis -- drug effects
- Caspase 3 -- metabolism
- Caspase 9 -- metabolism
- Cell Hypoxia
- Cell Line, Tumor
- Cell Survival -- drug effects
- Cytochromes c -- metabolism
- Cytoprotection
- Dose-Response Relationship, Drug
- Glucose -- deficiency
- Hydrogen Peroxide -- metabolism
- Membrane Potential, Mitochondrial -- drug effects
- Mice
- Mitochondria -- drug effects
- Neuroblastoma -- metabolism
- PPAR gamma -- agonists
- Phosphorylation
- Poly(ADP-ribose) Polymerases -- metabolism
- Protein Serine-Threonine Kinases -- metabolism
- Proto-Oncogene Proteins c-akt -- metabolism
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- RNA Interference
- RNA, Small Interfering
- Reperfusion Injury -- metabolism
- Rosiglitazone
- Thiazolidinediones -- pharmacology
- Time Factors
- Transfection
- Up-Regulation
- bcl-Associated Death Protein -- metabolism
- bcl-X Protein -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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