Apoptosis in experimental NASH is associated with p53 activation and TRAIL receptor expression. [electronic resource]
Producer: 20090625Description: 443-52 p. digitalISSN:- 1440-1746
- Alanine Transaminase -- blood
- Animals
- Apoptosis
- BH3 Interacting Domain Death Agonist Protein -- metabolism
- Caspase 3 -- metabolism
- Choline Deficiency -- complications
- Cyclin-Dependent Kinase Inhibitor p21 -- metabolism
- Disease Models, Animal
- Fatty Liver -- etiology
- Gene Expression Regulation
- Insulin-Like Growth Factor I -- metabolism
- Liver -- enzymology
- Male
- Methionine -- deficiency
- Mice
- Mice, Inbred C57BL
- Mitochondria, Liver -- enzymology
- Nutritional Status
- RNA, Messenger -- metabolism
- Receptors, TNF-Related Apoptosis-Inducing Ligand -- genetics
- Receptors, Tumor Necrosis Factor -- metabolism
- Receptors, Tumor Necrosis Factor, Type I -- metabolism
- Receptors, Tumor Necrosis Factor, Type II -- metabolism
- Time Factors
- Tumor Suppressor Protein p53 -- genetics
- bcl-X Protein -- metabolism
- fas Receptor -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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