Gene plasticity in colonic circular smooth muscle cells underlies motility dysfunction in a model of postinfective IBS. [electronic resource]
Producer: 20090428Description: G632-42 p. digitalISSN:- 0193-1857
- Acetylcholine -- pharmacology
- Age Factors
- Animals
- Animals, Newborn
- Calcium Channels, L-Type -- genetics
- Cholinergic Agents -- pharmacology
- Colon -- cytology
- Defecation -- physiology
- Disease Models, Animal
- Enteric Nervous System -- physiology
- Enteritis -- genetics
- GTP-Binding Protein alpha Subunits, Gq-G11 -- genetics
- Gastrointestinal Motility -- drug effects
- Gene Expression Regulation -- physiology
- Inflammation Mediators -- metabolism
- Irritable Bowel Syndrome -- genetics
- Male
- Myocytes, Smooth Muscle -- physiology
- Myosin Light Chains -- genetics
- Rats
- Rats, Sprague-Dawley
- Severity of Illness Index
- Signal Transduction -- physiology
- Vasoactive Intestinal Peptide -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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