Low-dose inhaled carbon monoxide attenuates the remote intestinal inflammatory response elicited by hindlimb ischemia-reperfusion. [electronic resource]
Producer: 20090226Description: G9-G14 p. digitalISSN:- 0193-1857
- Administration, Inhalation
- Animals
- Anti-Inflammatory Agents -- administration & dosage
- Carbon Monoxide -- administration & dosage
- Cytokines -- metabolism
- Disease Models, Animal
- Gastrointestinal Agents -- administration & dosage
- Hindlimb
- Ileitis -- etiology
- Interleukin-1beta -- metabolism
- Leukocyte Rolling -- drug effects
- Lipid Peroxidation -- drug effects
- Male
- Malondialdehyde -- metabolism
- Mice
- Mice, Inbred C57BL
- Microcirculation -- drug effects
- Muscle, Skeletal -- blood supply
- Oxidative Stress -- drug effects
- Reperfusion Injury -- complications
- Systemic Inflammatory Response Syndrome -- etiology
- Thiobarbituric Acid Reactive Substances -- metabolism
- Tumor Necrosis Factor-alpha -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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