The proapoptotic BCL-2 homology domain 3-only protein Bim is not critical for acute excitotoxic cell death. [electronic resource]
Producer: 20090409Description: 102-10 p. digitalISSN:- 0022-3069
- Animals
- Apoptosis -- drug effects
- Apoptosis Regulatory Proteins -- deficiency
- Bcl-2-Like Protein 11
- Caspase 3 -- metabolism
- DNA Fragmentation -- drug effects
- Disease Models, Animal
- Electroencephalography
- Excitatory Amino Acid Agonists -- toxicity
- Glial Fibrillary Acidic Protein -- metabolism
- Hippocampus -- drug effects
- In Situ Nick-End Labeling -- methods
- Kainic Acid -- toxicity
- Male
- Membrane Proteins -- deficiency
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Neurons -- drug effects
- Neurotoxicity Syndromes -- etiology
- Phosphopyruvate Hydratase -- metabolism
- Proto-Oncogene Proteins -- deficiency
- Seizures -- chemically induced
- Stereotaxic Techniques
- Time Factors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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