IL-10 inhibits inflammation and attenuates left ventricular remodeling after myocardial infarction via activation of STAT3 and suppression of HuR. [electronic resource]
Producer: 20090212Description: e9-18 p. digitalISSN:- 1524-4571
- Animals
- Anti-Inflammatory Agents -- administration & dosage
- Antigens, Surface -- genetics
- Apoptosis
- Arterioles -- metabolism
- Capillaries -- metabolism
- Disease Models, Animal
- ELAV Proteins
- ELAV-Like Protein 1
- Fibrosis
- Inflammation -- metabolism
- Inflammation Mediators -- metabolism
- Interleukin-10 -- administration & dosage
- Matrix Metalloproteinase 9 -- genetics
- Mice
- Mice, Inbred C57BL
- Myocardial Infarction -- metabolism
- Myocardium -- metabolism
- NIH 3T3 Cells
- Neovascularization, Physiologic
- Phosphorylation
- RNA Interference
- RNA, Messenger -- metabolism
- RNA, Small Interfering -- metabolism
- RNA-Binding Proteins -- genetics
- Recombinant Proteins -- metabolism
- STAT3 Transcription Factor -- metabolism
- Time Factors
- Vascular Endothelial Growth Factor A -- metabolism
- Ventricular Function, Left -- drug effects
- Ventricular Remodeling -- drug effects
- p38 Mitogen-Activated Protein Kinases -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.