Cross-linking of B7-H1 on EBV-transformed B cells induces apoptosis through reactive oxygen species production, JNK signaling activation, and fasL expression. [electronic resource]
Producer: 20090121Description: 6158-69 p. digitalISSN:- 1550-6606
- Animals
- Antibodies, Monoclonal -- metabolism
- Apoptosis -- immunology
- B-Lymphocyte Subsets -- enzymology
- B7-1 Antigen -- metabolism
- Callithrix
- Cell Line, Transformed
- Cross-Linking Reagents -- metabolism
- Enzyme Activation -- immunology
- Fas Ligand Protein -- biosynthesis
- Herpesvirus 4, Human -- immunology
- Humans
- Inducible T-Cell Co-Stimulator Ligand
- JNK Mitogen-Activated Protein Kinases -- metabolism
- MAP Kinase Signaling System -- immunology
- Mitogen-Activated Protein Kinase 1 -- antagonists & inhibitors
- Mitogen-Activated Protein Kinase 3 -- antagonists & inhibitors
- Phosphorylation -- immunology
- Proto-Oncogene Proteins c-akt -- antagonists & inhibitors
- Reactive Oxygen Species -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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