Activation of the PGI(2)/IP system contributes to the development of circulatory failure in a rat model of endotoxic shock. [electronic resource]
Producer: 20080822Description: 330-5 p. digitalISSN:- 1524-4563
- Analysis of Variance
- Animals
- Blotting, Western
- Cells, Cultured
- Cytokines -- metabolism
- Epoprostenol -- metabolism
- Iloprost -- pharmacology
- Lipopolysaccharides -- pharmacology
- Male
- Models, Animal
- Multiple Organ Failure -- physiopathology
- Muscle, Smooth, Vascular -- cytology
- Probability
- Prostaglandins F -- metabolism
- Prostaglandins I -- analysis
- RNA, Messenger -- analysis
- Random Allocation
- Rats
- Reverse Transcriptase Polymerase Chain Reaction
- Sensitivity and Specificity
- Shock, Septic -- physiopathology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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