Nuclear proteasomal degradation and cytoplasmic retention underlie early nuclear exclusion of transcription factor Max upon axon damage. [electronic resource]
Producer: 20080915Description: 202-9 p. digitalISSN:- 1090-2430
- Active Transport, Cell Nucleus -- genetics
- Animals
- Apoptosis -- genetics
- Axons -- drug effects
- Basic Helix-Loop-Helix Leucine Zipper Transcription Factors -- genetics
- Cell Nucleus -- drug effects
- Cytoplasm -- metabolism
- Dipeptides -- pharmacology
- Enzyme Inhibitors -- pharmacology
- Fatty Acids, Unsaturated -- pharmacology
- Karyopherins -- antagonists & inhibitors
- Organ Culture Techniques
- Proteasome Endopeptidase Complex -- genetics
- Protein Transport -- genetics
- Rats
- Receptors, Cytoplasmic and Nuclear -- antagonists & inhibitors
- Retinal Ganglion Cells -- drug effects
- Wallerian Degeneration -- metabolism
- Exportin 1 Protein
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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