Lack of inducible nitric oxide synthase leads to increased hepatic apoptosis and decreased fibrosis in mice after chronic carbon tetrachloride administration. [electronic resource]
Producer: 20080703Description: 2051-8 p. digitalISSN:- 1527-3350
- Animals
- Apoptosis -- drug effects
- Carbon Tetrachloride -- adverse effects
- Collagen -- genetics
- Liver -- enzymology
- Liver Cirrhosis -- chemically induced
- Male
- Malondialdehyde -- metabolism
- Matrix Metalloproteinase 2 -- genetics
- Matrix Metalloproteinase 9 -- genetics
- Mice
- Mice, Knockout
- Nitric Oxide Synthase Type II -- genetics
- Peroxynitrous Acid -- metabolism
- RNA, Messenger -- metabolism
- Reactive Oxygen Species -- metabolism
- Tissue Inhibitor of Metalloproteinase-1 -- metabolism
- Transaminases -- blood
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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