APA

Heath K. E., Argente J., Barrios V., Pozo J., Díaz-González F., Martos-Moreno G. A., Caimari M., Gracia R. & Campos-Barros A. (20080617). Primary acid-labile subunit deficiency due to recessive IGFALS mutations results in postnatal growth deficit associated with low circulating insulin growth factor (IGF)-I, IGF binding protein-3 levels, and hyperinsulinemia. : The Journal of clinical endocrinology and metabolism.

Chicago

Heath Karen E, Argente Jesús, Barrios Vicente, Pozo Jesús, Díaz-González Francisca, Martos-Moreno Gabriel A, Caimari María, Gracia Ricardo and Campos-Barros Angel. 20080617. Primary acid-labile subunit deficiency due to recessive IGFALS mutations results in postnatal growth deficit associated with low circulating insulin growth factor (IGF)-I, IGF binding protein-3 levels, and hyperinsulinemia. : The Journal of clinical endocrinology and metabolism.

Harvard

Heath K. E., Argente J., Barrios V., Pozo J., Díaz-González F., Martos-Moreno G. A., Caimari M., Gracia R. and Campos-Barros A. (20080617). Primary acid-labile subunit deficiency due to recessive IGFALS mutations results in postnatal growth deficit associated with low circulating insulin growth factor (IGF)-I, IGF binding protein-3 levels, and hyperinsulinemia. : The Journal of clinical endocrinology and metabolism.

MLA

Heath Karen E, Argente Jesús, Barrios Vicente, Pozo Jesús, Díaz-González Francisca, Martos-Moreno Gabriel A, Caimari María, Gracia Ricardo and Campos-Barros Angel. Primary acid-labile subunit deficiency due to recessive IGFALS mutations results in postnatal growth deficit associated with low circulating insulin growth factor (IGF)-I, IGF binding protein-3 levels, and hyperinsulinemia. : The Journal of clinical endocrinology and metabolism. 20080617.