Complement inhibition reduces injury in the type 2 diabetic heart following ischemia and reperfusion. [electronic resource]
Producer: 20080515Description: H1282-90 p. digitalISSN:- 0363-6135
- Animals
- Benzamidines
- Blood Cell Count
- Blood Glucose -- metabolism
- Blood Pressure -- physiology
- CD11b Antigen -- biosynthesis
- Cell Adhesion Molecules -- biosynthesis
- Cell Death -- physiology
- Complement Activation -- drug effects
- Complement C3 -- metabolism
- Complement Inactivating Agents -- therapeutic use
- Complement System Proteins -- metabolism
- Diabetes Mellitus, Type 2 -- genetics
- Diabetic Angiopathies -- pathology
- Flow Cytometry
- Guanidines -- pharmacology
- Heart Rate -- physiology
- Intercellular Adhesion Molecule-1 -- biosynthesis
- Male
- Myocardial Infarction -- pathology
- Myocardial Reperfusion Injury -- pathology
- Neutrophils -- metabolism
- Rats
- Rats, Zucker
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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