Sequestration of mutated alpha1-antitrypsin into inclusion bodies is a cell-protective mechanism to maintain endoplasmic reticulum function. [electronic resource]
Producer: 20081110Description: 572-86 p. digitalISSN:- 1939-4586
- Animals
- Autophagy
- Biomarkers -- metabolism
- Calnexin -- metabolism
- Carcinoma, Hepatocellular -- metabolism
- Cell Line, Tumor
- Cell Size
- Cytoprotection
- Endoplasmic Reticulum -- metabolism
- Humans
- Inclusion Bodies -- enzymology
- Lysosomes -- metabolism
- Mice
- Mutant Proteins -- metabolism
- Neurites -- metabolism
- Neuroblastoma -- metabolism
- Neuropeptides -- metabolism
- Phagosomes -- metabolism
- Protein Disulfide-Isomerases -- metabolism
- Protein Folding
- Protein Transport
- Serpins -- metabolism
- Time Factors
- alpha 1-Antitrypsin -- metabolism
- Neuroserpin
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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