NKG2D ligand expression in AML increases in response to HDAC inhibitor valproic acid and contributes to allorecognition by NK-cell lines with single KIR-HLA class I specificities. [electronic resource]
Producer: 20080326Description: 1428-36 p. digitalISSN:- 0006-4971
- Cell Line
- Cell Survival
- Cytotoxicity, Immunologic -- immunology
- Enzyme Inhibitors -- pharmacology
- GPI-Linked Proteins
- HLA Antigens -- immunology
- Histocompatibility Antigens Class I -- immunology
- Histone Deacetylase Inhibitors
- Histone Deacetylases -- metabolism
- Humans
- Intracellular Signaling Peptides and Proteins -- metabolism
- Killer Cells, Natural -- drug effects
- Leukemia, Myeloid, Acute -- blood
- Ligands
- Membrane Proteins -- metabolism
- NK Cell Lectin-Like Receptor Subfamily K
- Receptors, Immunologic -- metabolism
- Receptors, Natural Killer Cell
- Sensitivity and Specificity
- Solubility
- Up-Regulation -- drug effects
- Valproic Acid -- analogs & derivatives
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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