Oxidative stress and adenosine A1 receptor activation differentially modulate subcellular cardiomyocyte MAPKs. [electronic resource]
Producer: 20080228Description: H263-71 p. digitalISSN:- 0363-6135
- Adenosine -- analogs & derivatives
- Adenosine A1 Receptor Agonists
- Animals
- Antioxidants -- pharmacology
- Caveolin 3 -- metabolism
- Cell Membrane -- enzymology
- Cell Nucleus -- enzymology
- Cytosol -- enzymology
- Detergents
- Enzyme Activation
- Hydrogen Peroxide -- pharmacology
- In Vitro Techniques
- MAP Kinase Signaling System -- drug effects
- Male
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinase 14 -- metabolism
- Mitogen-Activated Protein Kinase 3 -- metabolism
- Mitogen-Activated Protein Kinases -- metabolism
- Myocytes, Cardiac -- drug effects
- Octoxynol
- Oxidants -- pharmacology
- Oxidation-Reduction
- Oxidative Stress -- drug effects
- Protein Phosphatase 2 -- metabolism
- Rats
- Rats, Sprague-Dawley
- Reactive Oxygen Species -- metabolism
- Receptor, Adenosine A1 -- metabolism
- Subcellular Fractions -- enzymology
- Tiopronin -- pharmacology
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Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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