Glucocorticoids regulate innate immunity in a model of multiple sclerosis: reciprocal interactions between the A1 adenosine receptor and beta-arrestin-1 in monocytoid cells. [electronic resource]
Producer: 20080410Description: 786-96 p. digitalISSN:- 1530-6860
- Animals
- Arrestins -- metabolism
- Brain -- metabolism
- Cells, Cultured
- Disease Models, Animal
- Down-Regulation
- Encephalomyelitis, Autoimmune, Experimental -- immunology
- Glucocorticoids -- metabolism
- Humans
- Immunity, Innate -- drug effects
- Mice
- Monocytes -- immunology
- Multiple Sclerosis -- immunology
- Receptor, Adenosine A1 -- drug effects
- beta-Arrestin 1
- beta-Arrestins
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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