Type 1 diabetes and hypercholesterolaemia reveal the contribution of endothelium-derived hyperpolarizing factor to endothelium-dependent relaxation of the rat aorta. [electronic resource]
Producer: 20080131Description: 192-200 p. digitalISSN:- 1440-1681
- Acetylcholine -- pharmacology
- Animals
- Aorta, Thoracic -- drug effects
- Apamin -- pharmacology
- Biological Factors -- metabolism
- Blood Glucose -- metabolism
- Body Weight
- Charybdotoxin -- pharmacology
- Cholesterol -- blood
- Cyclooxygenase Inhibitors -- pharmacology
- Diabetes Mellitus, Experimental -- metabolism
- Diabetes Mellitus, Type 1 -- metabolism
- Disease Models, Animal
- Dose-Response Relationship, Drug
- Enzyme Inhibitors -- pharmacology
- Guanylate Cyclase -- antagonists & inhibitors
- Hypercholesterolemia -- metabolism
- Indomethacin -- pharmacology
- Male
- Nitric Oxide -- metabolism
- Nitric Oxide Synthase -- antagonists & inhibitors
- Nitroarginine -- pharmacology
- Nitroprusside -- pharmacology
- Oxadiazoles -- pharmacology
- Oxidative Stress
- Potassium Channel Blockers -- pharmacology
- Potassium Channels, Calcium-Activated -- antagonists & inhibitors
- Quinoxalines -- pharmacology
- Rats
- Rats, Sprague-Dawley
- Receptors, Cytoplasmic and Nuclear -- antagonists & inhibitors
- Soluble Guanylyl Cyclase
- Superoxides -- metabolism
- Vasodilation -- drug effects
- Vasodilator Agents -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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