IL-17 stimulates MMP-1 expression in primary human cardiac fibroblasts via p38 MAPK- and ERK1/2-dependent C/EBP-beta , NF-kappaB, and AP-1 activation. [electronic resource]
Producer: 20080131Description: H3356-65 p. digitalISSN:- 0363-6135
- CCAAT-Enhancer-Binding Protein-beta -- genetics
- Cell Movement
- Cells, Cultured
- Enzyme Induction
- Extracellular Signal-Regulated MAP Kinases -- antagonists & inhibitors
- Fibroblasts -- drug effects
- Genes, Reporter
- Humans
- Interleukin-17 -- metabolism
- Matrix Metalloproteinase 1 -- biosynthesis
- Matrix Metalloproteinase 13 -- biosynthesis
- Matrix Metalloproteinase 2 -- biosynthesis
- Matrix Metalloproteinase 3 -- biosynthesis
- Matrix Metalloproteinase 9 -- biosynthesis
- Matrix Metalloproteinase Inhibitors
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinase 3 -- metabolism
- Myocardium -- enzymology
- NF-kappa B -- genetics
- Oligonucleotides, Antisense -- metabolism
- Promoter Regions, Genetic
- Protease Inhibitors -- pharmacology
- Protein Kinase Inhibitors -- pharmacology
- RNA Interference
- RNA, Messenger -- biosynthesis
- RNA, Small Interfering -- metabolism
- Recombinant Proteins -- metabolism
- Signal Transduction
- Tissue Inhibitor of Metalloproteinase-1 -- biosynthesis
- Transcription Factor AP-1 -- metabolism
- Transcription, Genetic
- Transfection
- p38 Mitogen-Activated Protein Kinases -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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