APA

Kobayashi J., Nishida T., Ameis D., Stahnke G., Schotz M. C., Hashimoto H., Fukamachi I., Shirai K., Saito Y. & Yoshida S. (19920218). A heterozygous mutation (the codon for Ser447----a stop codon) in lipoprotein lipase contributes to a defect in lipid interface recognition in a case with type I hyperlipidemia. : Biochemical and biophysical research communications.

Chicago

Kobayashi J, Nishida T, Ameis D, Stahnke G, Schotz M C, Hashimoto H, Fukamachi I, Shirai K, Saito Y and Yoshida S. 19920218. A heterozygous mutation (the codon for Ser447----a stop codon) in lipoprotein lipase contributes to a defect in lipid interface recognition in a case with type I hyperlipidemia. : Biochemical and biophysical research communications.

Harvard

Kobayashi J., Nishida T., Ameis D., Stahnke G., Schotz M. C., Hashimoto H., Fukamachi I., Shirai K., Saito Y. and Yoshida S. (19920218). A heterozygous mutation (the codon for Ser447----a stop codon) in lipoprotein lipase contributes to a defect in lipid interface recognition in a case with type I hyperlipidemia. : Biochemical and biophysical research communications.

MLA

Kobayashi J, Nishida T, Ameis D, Stahnke G, Schotz M C, Hashimoto H, Fukamachi I, Shirai K, Saito Y and Yoshida S. A heterozygous mutation (the codon for Ser447----a stop codon) in lipoprotein lipase contributes to a defect in lipid interface recognition in a case with type I hyperlipidemia. : Biochemical and biophysical research communications. 19920218.