Akt-induced endocrine therapy resistance is reversed by inhibition of mTOR signaling. [electronic resource]
Producer: 20071206Description: 1323-8 p. digitalISSN:- 0923-7534
- Antineoplastic Agents, Hormonal -- pharmacology
- Antineoplastic Combined Chemotherapy Protocols -- pharmacology
- Apoptosis -- drug effects
- Aromatase Inhibitors -- pharmacology
- Blotting, Western
- Breast Neoplasms -- drug therapy
- Cell Line, Tumor
- Cell Proliferation -- drug effects
- Drug Resistance, Neoplasm -- physiology
- Estradiol -- analogs & derivatives
- Everolimus
- Female
- Flow Cytometry
- Fulvestrant
- Humans
- Letrozole
- Nitriles -- pharmacology
- Protein Kinases -- drug effects
- Proto-Oncogene Proteins c-akt -- metabolism
- Sirolimus -- analogs & derivatives
- TOR Serine-Threonine Kinases
- Triazoles -- pharmacology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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