Cardioprotection induced by hydrogen sulfide preconditioning involves activation of ERK and PI3K/Akt pathways. [electronic resource]
Producer: 20080612Description: 607-16 p. digitalISSN:- 0031-6768
- Animals
- Benzophenanthridines -- pharmacology
- Cardiotonic Agents -- metabolism
- Cell Survival -- drug effects
- Chromones -- pharmacology
- Disease Models, Animal
- Dose-Response Relationship, Drug
- Enzyme Activation
- Extracellular Signal-Regulated MAP Kinases -- antagonists & inhibitors
- Flavonoids -- pharmacology
- Glyburide -- pharmacology
- Hydrogen Sulfide -- metabolism
- Ischemic Preconditioning, Myocardial
- KATP Channels -- antagonists & inhibitors
- Male
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinase 3 -- metabolism
- Morpholines -- pharmacology
- Myocardial Contraction -- drug effects
- Myocardial Infarction -- metabolism
- Myocardial Reperfusion Injury -- metabolism
- Myocardium -- enzymology
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphoinositide-3 Kinase Inhibitors
- Phosphorylation
- Potassium Channel Blockers -- pharmacology
- Protein Kinase C -- antagonists & inhibitors
- Protein Kinase Inhibitors -- pharmacology
- Proto-Oncogene Proteins c-akt -- antagonists & inhibitors
- Rats
- Rats, Sprague-Dawley
- Signal Transduction -- drug effects
- Sulfides -- metabolism
- Time Factors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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