Constitutively active calcineurin mediates delayed neuronal death through Fas-ligand expression via activation of NFAT and FKHR transcriptional activities in mouse brain ischemia. [electronic resource]
Producer: 20071106Description: 1506-1517 p. digitalISSN:- 0022-3042
- Analysis of Variance
- Animals
- Brain Ischemia -- drug therapy
- Calcineurin -- metabolism
- Cell Death -- drug effects
- Electrophoretic Mobility Shift Assay -- methods
- Enzyme Activation -- drug effects
- Fas Ligand Protein -- metabolism
- Forkhead Box Protein O1
- Forkhead Transcription Factors -- metabolism
- Gene Expression Regulation -- drug effects
- Immunoprecipitation -- methods
- Immunosuppressive Agents -- pharmacology
- Male
- Mice
- Mice, Inbred C57BL
- NFATC Transcription Factors -- metabolism
- Oncogene Protein v-akt -- metabolism
- Phosphorylation -- drug effects
- Protein Transport -- drug effects
- Serine -- metabolism
- Tacrolimus -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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