Loss of FANCC function is associated with failure to inhibit late firing replication origins after DNA cross-linking. [electronic resource]
Producer: 20070817Description: 2283-92 p. digitalISSN:- 0014-4827
- Cell Cycle -- drug effects
- Cell Line
- Cross-Linking Reagents -- toxicity
- DNA -- drug effects
- DNA Damage -- genetics
- DNA Repair -- genetics
- DNA Replication -- genetics
- Epoxy Compounds -- toxicity
- Fanconi Anemia -- genetics
- Fanconi Anemia Complementation Group A Protein -- genetics
- Fanconi Anemia Complementation Group C Protein -- genetics
- Fanconi Anemia Complementation Group D2 Protein -- genetics
- Humans
- Oncogene Proteins, Viral -- genetics
- Replication Origin -- genetics
- Repressor Proteins -- genetics
- S Phase -- drug effects
- Tumor Suppressor Protein p53 -- metabolism
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Publication Type: Journal Article
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