Apigenin-induced cell cycle arrest is mediated by modulation of MAPK, PI3K-Akt, and loss of cyclin D1 associated retinoblastoma dephosphorylation in human prostate cancer cells. [electronic resource]
Producer: 20070807Description: 1102-14 p. digitalISSN:- 1551-4005
- Antineoplastic Agents, Phytogenic -- pharmacology
- Apigenin -- pharmacology
- Cell Cycle -- drug effects
- Cyclin D1 -- metabolism
- Humans
- MAP Kinase Signaling System -- drug effects
- Male
- Mitogen-Activated Protein Kinases -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphorylation -- drug effects
- Prostatic Neoplasms -- enzymology
- Proto-Oncogene Proteins c-akt -- metabolism
- Retinoblastoma Protein -- metabolism
- Tumor Cells, Cultured
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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