Selective dysfunction of hippocampal CA1 astrocytes contributes to delayed neuronal damage after transient forebrain ischemia. [electronic resource]
Producer: 20070507Description: 4253-60 p. digitalISSN:- 1529-2401
- Animals
- Astrocytes -- metabolism
- Ceftriaxone -- pharmacology
- Cell Survival
- Excitatory Amino Acid Transporter 2 -- metabolism
- Glial Fibrillary Acidic Protein -- metabolism
- Glutamic Acid -- pharmacokinetics
- Hippocampus -- physiopathology
- Immunohistochemistry
- Ischemic Attack, Transient -- physiopathology
- Male
- Membrane Potentials
- Mitochondria -- metabolism
- Oxidative Stress
- Rats
- Rats, Sprague-Dawley
- Up-Regulation
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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