RIP1-mediated AIP1 phosphorylation at a 14-3-3-binding site is critical for tumor necrosis factor-induced ASK1-JNK/p38 activation. [electronic resource]
Producer: 20070718Description: 14788-96 p. digitalISSN:- 0021-9258
- 14-3-3 Proteins -- metabolism
- Adaptor Proteins, Signal Transducing
- Amino Acid Substitution
- Animals
- Apoptosis -- drug effects
- Carrier Proteins
- Cattle
- Cells, Cultured
- Endothelial Cells -- cytology
- Enzyme Activation -- drug effects
- Guanylate Kinases
- Humans
- MAP Kinase Kinase 4 -- metabolism
- MAP Kinase Kinase Kinase 5 -- metabolism
- MAP Kinase Signaling System -- drug effects
- Multiprotein Complexes -- genetics
- Mutation, Missense
- Phosphorylation -- drug effects
- Protein Binding -- drug effects
- Protein Processing, Post-Translational -- drug effects
- Proteins -- genetics
- Receptor-Interacting Protein Serine-Threonine Kinases -- metabolism
- TNF Receptor-Associated Factor 2 -- metabolism
- Tumor Necrosis Factor-alpha -- metabolism
- p38 Mitogen-Activated Protein Kinases -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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