Targeting homeostatic mechanisms of endoplasmic reticulum stress to increase susceptibility of cancer cells to fenretinide-induced apoptosis: the role of stress proteins ERdj5 and ERp57. [electronic resource]
Producer: 20070531Description: 1062-71 p. digitalISSN:- 0007-0920
- Activating Transcription Factor 4 -- genetics
- Alternative Splicing
- Antineoplastic Agents -- pharmacology
- Apoptosis -- drug effects
- Biomarkers, Tumor -- metabolism
- DNA-Binding Proteins -- genetics
- Endoplasmic Reticulum -- metabolism
- Endoplasmic Reticulum Chaperone BiP
- Eukaryotic Initiation Factor-2 -- genetics
- Fenretinide -- pharmacology
- HSP40 Heat-Shock Proteins
- Humans
- Molecular Chaperones -- antagonists & inhibitors
- Neuroblastoma -- drug therapy
- Neuroectodermal Tumors -- drug therapy
- Nuclear Proteins -- genetics
- Oligonucleotide Array Sequence Analysis
- Oxidative Stress
- Phosphorylation -- drug effects
- Protein Disulfide-Isomerases -- antagonists & inhibitors
- RNA, Messenger -- genetics
- RNA, Small Interfering -- pharmacology
- Reactive Oxygen Species -- metabolism
- Regulatory Factor X Transcription Factors
- Transcription Factors
- Tumor Cells, Cultured -- drug effects
- X-Box Binding Protein 1
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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