A common SCN5A variant alters the responsiveness of human sodium channels to class I antiarrhythmic agents. [electronic resource]
Producer: 20070607Description: 434-40 p. digitalISSN:- 1540-8167
- Anti-Arrhythmia Agents -- classification
- Cells, Cultured
- Flecainide -- pharmacokinetics
- Genetic Variation -- genetics
- Humans
- Ion Channel Gating -- drug effects
- Mexiletine -- pharmacokinetics
- Muscle Proteins -- genetics
- NAV1.5 Voltage-Gated Sodium Channel
- Patch-Clamp Techniques
- Quinidine -- pharmacokinetics
- Sodium Channels -- drug effects
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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