Targeting cellular prion protein reverses early cognitive deficits and neurophysiological dysfunction in prion-infected mice. [electronic resource]
Producer: 20070315Description: 325-35 p. digitalISSN:- 0896-6273
- Animals
- Axons -- physiology
- Behavior, Animal -- physiology
- Brain -- pathology
- Cognition Disorders -- prevention & control
- Discrimination, Psychological -- physiology
- Electrophysiology
- Hippocampus -- pathology
- Immunohistochemistry
- Long-Term Potentiation -- physiology
- Memory Disorders -- etiology
- Mice
- Mice, Transgenic
- Motor Activity -- physiology
- Muscle, Skeletal -- physiology
- Nesting Behavior -- physiology
- PrPC Proteins -- genetics
- Prion Diseases -- genetics
- Psychomotor Performance -- physiology
- Reverse Transcriptase Polymerase Chain Reaction
- Synapses -- pathology
- Visual Perception -- physiology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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