Rescue of neurological deficits in a mouse model for Angelman syndrome by reduction of alphaCaMKII inhibitory phosphorylation. [electronic resource]
Producer: 20070516Description: 280-2 p. digitalISSN:- 1097-6256
- Angelman Syndrome -- complications
- Animals
- Behavior, Animal
- Calcium-Calmodulin-Dependent Protein Kinase Type 2 -- genetics
- Conditioning, Classical -- physiology
- Disease Models, Animal
- Excitatory Postsynaptic Potentials -- genetics
- Female
- Freezing Reaction, Cataleptic -- physiology
- Hippocampus -- physiopathology
- In Vitro Techniques
- Male
- Maze Learning -- physiology
- Mental Disorders -- etiology
- Mice
- Mice, Inbred C57BL
- Mice, Neurologic Mutants
- Motor Activity -- physiology
- Phosphorylation
- Phosphotransferases -- metabolism
- Reaction Time
- Time Factors
- Ubiquitin-Protein Ligases -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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