HPV integration begins in the tonsillar crypt and leads to the alteration of p16, EGFR and c-myc during tumor formation. [electronic resource]
Producer: 20070403Description: 1418-25 p. digitalISSN:- 0020-7136
- Carcinoma, Squamous Cell -- metabolism
- Cyclin A -- genetics
- Cyclin A1
- Cyclin B -- genetics
- Cyclin B1
- Cyclin-Dependent Kinase Inhibitor p16 -- genetics
- DNA, Viral -- genetics
- ErbB Receptors -- genetics
- Gene Amplification
- Humans
- Hypoxia-Inducible Factor 1, alpha Subunit -- genetics
- In Situ Hybridization, Fluorescence
- Inhibitor of Apoptosis Proteins
- Microtubule-Associated Proteins -- genetics
- Neoplasm Proteins -- genetics
- Papillomaviridae -- physiology
- Proto-Oncogene Proteins c-myc -- genetics
- RNA, Messenger -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- S-Phase Kinase-Associated Proteins -- genetics
- Survivin
- Tissue Array Analysis
- Tonsillar Neoplasms -- metabolism
- Tonsillitis -- genetics
- Virus Integration -- physiology
No physical items for this record
Publication Type: Journal Article
There are no comments on this title.
Log in to your account to post a comment.