Elevated endogenous nitric oxide increases Ca2+ flux via L-type Ca2+ channels by S-nitrosylation in rat hippocampal neurons during severe hypoxia and in vitro ischemia. [electronic resource]
Producer: 20070215Description: 52-63 p. digitalISSN:- 0891-5849
- Animals
- Arginine -- pharmacology
- Brain Ischemia -- metabolism
- Calcium -- metabolism
- Calcium Channels, L-Type -- metabolism
- Cell Hypoxia
- Cells, Cultured
- Cyclic GMP -- metabolism
- Electrophysiology
- Enzyme Inhibitors -- pharmacology
- Fura-2
- Hippocampus -- cytology
- NG-Nitroarginine Methyl Ester -- pharmacology
- Neurons -- cytology
- Nitric Oxide -- pharmacology
- Nitric Oxide Synthase -- antagonists & inhibitors
- Oxygen -- metabolism
- Rats
- Rats, Sprague-Dawley
- S-Nitrosothiols -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.