Increased oxygen radical formation and mitochondrial dysfunction mediate beta cell apoptosis under conditions of AMP-activated protein kinase stimulation. [electronic resource]
Producer: 20070215Description: 64-78 p. digitalISSN:- 0891-5849
- AMP-Activated Protein Kinases
- Aminoimidazole Carboxamide -- analogs & derivatives
- Animals
- Apoptosis -- physiology
- Blotting, Western
- Caspase 3 -- metabolism
- Cell Line
- Enzyme Activation
- Enzyme Inhibitors -- pharmacology
- Glucose -- pharmacology
- Hydrogen Peroxide -- pharmacology
- Hypoglycemic Agents -- metabolism
- Insulin -- metabolism
- Insulin-Secreting Cells -- metabolism
- JNK Mitogen-Activated Protein Kinases -- metabolism
- Mice
- Mitochondria -- metabolism
- Multienzyme Complexes -- metabolism
- Palmitates -- pharmacology
- Phosphorylation
- Protein Serine-Threonine Kinases -- metabolism
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- RNA, Messenger -- genetics
- Rats
- Reactive Oxygen Species -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- Ribonucleotides -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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