R5- and X4-HIV-1 use differentially the endometrial epithelial cells HEC-1A to ensure their own spread: implication for mechanisms of sexual transmission. [electronic resource]
Producer: 20070319Description: 55-68 p. digitalISSN:- 0042-6822
- Acetylglucosamine -- pharmacology
- Anti-HIV Agents -- pharmacology
- CCR5 Receptor Antagonists
- Cell Line
- Chemokine CXCL1
- Chemokines, CXC -- biosynthesis
- Endocytosis -- drug effects
- Endometrium -- virology
- Epithelial Cells -- virology
- Female
- Fucose -- pharmacology
- HIV Core Protein p24 -- analysis
- HIV Infections -- transmission
- HIV-1 -- immunology
- Heparan Sulfate Proteoglycans -- physiology
- Humans
- Interleukin-1alpha -- biosynthesis
- Interleukin-8 -- biosynthesis
- Lectins, C-Type -- physiology
- Mannose -- analogs & derivatives
- Mannose Receptor
- Mannose-Binding Lectins -- physiology
- Receptors, CCR5 -- physiology
- Receptors, CXCR4 -- physiology
- Receptors, Cell Surface -- physiology
- Tumor Necrosis Factor-alpha -- biosynthesis
- Virulence
- Virus Attachment
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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