NMDA receptor-nitric oxide transmission mediates neuronal iron homeostasis via the GTPase Dexras1. [electronic resource]
Producer: 20060926Description: 431-40 p. digitalISSN:- 0896-6273
- Adaptor Proteins, Signal Transducing
- Aldehydes -- pharmacology
- Animals
- Blotting, Western -- methods
- Cation Transport Proteins -- metabolism
- Cells, Cultured
- Cerebral Cortex -- cytology
- Dizocilpine Maleate -- pharmacology
- Dose-Response Relationship, Drug
- Drug Interactions
- Embryo, Mammalian
- Excitatory Amino Acid Agents -- pharmacology
- Fluorescent Antibody Technique -- methods
- Homeostasis -- drug effects
- Humans
- Hydrazones -- pharmacology
- Immunoprecipitation -- methods
- Iron -- metabolism
- Iron Chelating Agents -- pharmacology
- Iron-Binding Proteins -- metabolism
- Membrane Proteins
- Mice
- Mice, Knockout
- Models, Biological
- N-Methylaspartate -- pharmacology
- Neurons -- drug effects
- Nitric Oxide -- physiology
- Nitric Oxide Synthase Type I -- deficiency
- Protein Binding -- drug effects
- Rats
- Reactive Oxygen Species -- immunology
- Receptors, GABA-A -- metabolism
- Receptors, N-Methyl-D-Aspartate -- physiology
- Signal Transduction -- drug effects
- Transfection -- methods
- ras Proteins -- physiology
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Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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