Cardioprotection via activation of protein kinase C-delta depends on modulation of the reverse mode of the Na+/Ca2+ exchanger. [electronic resource]
Producer: 20060818Description: I226-32 p. digitalISSN:- 1524-4539
- Anesthetics, Inhalation -- pharmacology
- Aniline Compounds -- pharmacology
- Animals
- Calcium Signaling -- drug effects
- Cardiotonic Agents -- pharmacology
- Enzyme Activation
- Glucose -- pharmacology
- Heart Ventricles -- drug effects
- In Vitro Techniques
- Ischemic Preconditioning, Myocardial -- methods
- Male
- Methyl Ethers -- pharmacology
- Myocardial Ischemia -- enzymology
- Myocardial Reperfusion Injury -- enzymology
- Phenyl Ethers -- pharmacology
- Phosphorylation -- drug effects
- Phosphoserine -- analysis
- Protein Kinase C-delta -- physiology
- Protein Processing, Post-Translational -- drug effects
- Protein Transport -- drug effects
- Rats
- Rats, Wistar
- Sarcolemma -- enzymology
- Sevoflurane
- Signal Transduction -- drug effects
- Sodium-Calcium Exchanger -- antagonists & inhibitors
- Thiourea -- analogs & derivatives
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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