Pravastatin improves remodeling and cardiac function after myocardial infarction by an antiinflammatory mechanism rather than by the induction of angiogenesis. [electronic resource]
Producer: 20070927Description: 2217-25 p. digitalISSN:- 1552-6259
- Angiogenesis Inducing Agents -- pharmacology
- Animals
- Anti-Inflammatory Agents, Non-Steroidal -- pharmacology
- Body Weight -- drug effects
- Bone Marrow Cells -- drug effects
- Bone Marrow Transplantation
- Cell Differentiation -- drug effects
- Cells, Cultured -- drug effects
- Collagen -- analysis
- Combined Modality Therapy
- Drug Evaluation, Preclinical
- Endothelium, Vascular -- cytology
- Hematopoietic Stem Cell Mobilization
- Hematopoietic Stem Cell Transplantation
- Hydroxymethylglutaryl-CoA Reductase Inhibitors -- pharmacology
- Injections
- Interleukin-1beta -- metabolism
- Male
- Myocardial Infarction -- diagnostic imaging
- Neovascularization, Physiologic -- drug effects
- Pravastatin -- pharmacology
- Random Allocation
- Rats
- Rats, Zucker
- Single-Blind Method
- Triglycerides -- blood
- Ultrasonography
- Vascular Endothelial Growth Factor A -- metabolism
- Ventricular Remodeling -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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