Short amyloid-beta (Abeta) immunogens reduce cerebral Abeta load and learning deficits in an Alzheimer's disease mouse model in the absence of an Abeta-specific cellular immune response. [electronic resource]
Producer: 20060531Description: 4717-28 p. digitalISSN:- 1529-2401
- Alzheimer Disease -- complications
- Amyloid beta-Peptides -- administration & dosage
- Amyloid beta-Protein Precursor -- genetics
- Analysis of Variance
- Animals
- Antibodies -- blood
- Antibody Specificity
- Bacterial Toxins -- immunology
- Behavior, Animal
- Biophysical Phenomena
- Biophysics
- Brain Chemistry
- Cell Proliferation
- Cytokines -- metabolism
- Disease Models, Animal
- Enterotoxins -- immunology
- Enzyme-Linked Immunosorbent Assay -- methods
- Epitope Mapping -- methods
- Escherichia coli Proteins -- immunology
- Immunization, Secondary
- Immunohistochemistry -- methods
- Learning Disabilities -- etiology
- Male
- Maze Learning -- physiology
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Peptide Fragments -- administration & dosage
- T-Lymphocytes -- immunology
- Time Factors
- Vaccines
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Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural
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