Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCepsilon phosphorylation. [electronic resource]

By: Contributor(s): Producer: 20060613Description: 465-73 p. digitalISSN:
  • 0022-2828
Subject(s): Online resources: In: Journal of molecular and cellular cardiology vol. 40
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Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

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