Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCepsilon phosphorylation. [electronic resource]
Producer: 20060613Description: 465-73 p. digitalISSN:- 0022-2828
- Aging -- genetics
- Amino Acid Substitution
- Animals
- Cell Line
- Heart Failure -- genetics
- Mice
- Mice, Transgenic
- Myocardial Contraction -- genetics
- Myocardium -- metabolism
- Phosphorylation
- Protein Kinase C-epsilon -- metabolism
- Protein Processing, Post-Translational -- genetics
- Troponin I -- genetics
- Ventricular Function, Left -- genetics
No physical items for this record
Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.