Porphyromonas gingivalis-related cardiac cell apoptosis was majorly co-activated by p38 and extracellular signal-regulated kinase pathways. [electronic resource]
Producer: 20060323Description: 39-46 p. digitalISSN:- 0022-3484
- Animals
- Anthracenes -- pharmacology
- Apoptosis -- physiology
- Butadienes -- pharmacology
- Calcineurin Inhibitors
- Caspases -- physiology
- Cell Nucleus -- ultrastructure
- Chromones -- pharmacology
- Cyclosporine -- pharmacology
- DNA Fragmentation
- Enzyme Inhibitors -- pharmacology
- Fluorescent Dyes
- Imidazoles -- pharmacology
- Indoles
- MAP Kinase Kinase 4 -- antagonists & inhibitors
- Mitogen-Activated Protein Kinase 1 -- antagonists & inhibitors
- Mitogen-Activated Protein Kinase 3 -- antagonists & inhibitors
- Mitogen-Activated Protein Kinase Kinases -- antagonists & inhibitors
- Morpholines -- pharmacology
- Myocardium -- cytology
- Nitriles -- pharmacology
- Phosphoinositide-3 Kinase Inhibitors
- Porphyromonas gingivalis -- physiology
- Pyridines -- pharmacology
- Rats
- p38 Mitogen-Activated Protein Kinases -- antagonists & inhibitors
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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