The mitochondrial permeability transition pore and the Ca2+-activated K+ channel contribute to the cardioprotection conferred by tumor necrosis factor-alpha. [electronic resource]
Producer: 20060407Description: 199-205 p. digitalISSN:- 1043-4666
- Animals
- Atractyloside -- pharmacology
- Coronary Circulation -- physiology
- Cyclosporine -- pharmacology
- In Vitro Techniques
- Ion Channels -- drug effects
- L-Lactate Dehydrogenase -- biosynthesis
- Male
- Mitochondria, Heart -- drug effects
- Mitochondrial Membrane Transport Proteins
- Mitochondrial Permeability Transition Pore
- Myocardial Contraction -- drug effects
- Myocardial Infarction -- prevention & control
- Myocardial Ischemia -- drug therapy
- Myocardial Reperfusion Injury -- prevention & control
- Permeability
- Potassium Channel Blockers -- pharmacology
- Potassium Channels, Calcium-Activated -- drug effects
- Rats
- Rats, Sprague-Dawley
- Tumor Necrosis Factor-alpha -- pharmacology
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Publication Type: Journal Article
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