Transgenic disruption of gap junctional intercellular communication enhances early but not late stage hepatocarcinogenesis in the rat. [electronic resource]
Producer: 20060123Description: 695-701 p. digitalISSN:- 0192-6233
- Alkylating Agents
- Animals
- Animals, Genetically Modified
- Cell Communication -- drug effects
- Cocarcinogenesis
- Connexins -- analysis
- Diethylnitrosamine
- Disease Susceptibility
- Enzyme Induction
- Gap Junctions -- drug effects
- Glutathione Transferase -- biosynthesis
- Hepatectomy
- Liver Neoplasms, Experimental -- chemically induced
- Liver Regeneration -- physiology
- Male
- Mutation
- Phenobarbital -- administration & dosage
- Precancerous Conditions -- chemically induced
- Rats
- Time Factors
- Gap Junction beta-1 Protein
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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