Parenchymal regression in chronic pancreatitis spares islets reprogrammed for the expression of NFkappaB and IAPs. [electronic resource]
Producer: 20060112Description: 1263-75 p. digitalISSN:- 0023-6837
- Apoptosis
- Apoptosis Regulatory Proteins -- biosynthesis
- Cell Line, Tumor
- Epithelium -- metabolism
- Fibrosis
- Gene Expression Regulation
- Humans
- I-kappa B Kinase -- metabolism
- Inhibitor of Apoptosis Proteins -- biosynthesis
- Insulinoma
- Interferon-gamma -- physiology
- Islets of Langerhans -- metabolism
- Membrane Glycoproteins -- biosynthesis
- Microtubule-Associated Proteins -- biosynthesis
- NF-kappa B -- biosynthesis
- Neoplasm Proteins -- biosynthesis
- Pancreatitis, Chronic -- metabolism
- Protein Subunits -- biosynthesis
- Receptors, Tumor Necrosis Factor -- biosynthesis
- Survivin
- TNF-Related Apoptosis-Inducing Ligand
- Transcription Factor RelA -- biosynthesis
- Transforming Growth Factor beta -- physiology
- Tumor Necrosis Factor Decoy Receptors
- Tumor Necrosis Factor-alpha -- biosynthesis
- Up-Regulation
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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