Molecular control of physiological and pathological T-cell recruitment after mouse spinal cord injury. [electronic resource]
Producer: 20060221Description: 6576-83 p. digitalISSN:- 1529-2401
- Animals
- Chemokine CCL5 -- biosynthesis
- Chemokine CXCL10
- Chemokines -- biosynthesis
- Chemokines, CC -- biosynthesis
- Chemokines, CXC -- biosynthesis
- Chemotaxis, Leukocyte -- physiology
- Contusions -- immunology
- Genes, T-Cell Receptor beta
- Growth Substances -- biosynthesis
- Insulin-Like Growth Factor I -- biosynthesis
- Interleukin 1 Receptor Antagonist Protein
- Lymphocyte Activation -- immunology
- Lymphokines
- Macrophages -- physiology
- Mice
- Mice, Transgenic
- Microglia -- physiology
- Myelin Basic Protein -- immunology
- Myelitis -- etiology
- RNA, Messenger -- biosynthesis
- Sialoglycoproteins -- biosynthesis
- Spinal Cord Injuries -- immunology
- T-Cell Antigen Receptor Specificity
- T-Lymphocyte Subsets -- immunology
- Transforming Growth Factor beta -- biosynthesis
- Transforming Growth Factor beta1
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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