Bortezomib abolishes tumor necrosis factor-related apoptosis-inducing ligand resistance via a p21-dependent mechanism in human bladder and prostate cancer cells. [electronic resource]
Producer: 20050728Description: 4902-8 p. digitalISSN:- 0008-5472
- Apoptosis -- drug effects
- Apoptosis Regulatory Proteins
- Boronic Acids -- pharmacology
- Bortezomib
- Caspase 3
- Caspase 8
- Caspases -- metabolism
- Cell Cycle Proteins -- metabolism
- Cell Line, Tumor
- Cyclin-Dependent Kinase Inhibitor p21
- Drug Resistance, Neoplasm
- Drug Synergism
- Enzyme Activation -- drug effects
- Humans
- Male
- Membrane Glycoproteins -- pharmacology
- Prostatic Neoplasms -- drug therapy
- Pyrazines -- pharmacology
- TNF-Related Apoptosis-Inducing Ligand
- Tumor Necrosis Factor-alpha -- pharmacology
- Urinary Bladder Neoplasms -- drug therapy
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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